← SciSim / Biology

Mitosis

🧬 Tier: A-level / IB Biology & Early Undergraduate

Watch a single cell condense its chromosomes, line them up at the equator, pull sister chromatids apart, and split into two genetically identical daughter cells — the engine of growth, repair, and asexual reproduction.

1 · Interactive Simulation

🔬 Cell View (Engine B)
⏱ Cell Cycle Timeline
📊 DNA Content Graph
🆚 Mitosis vs Meiosis
Phase 1 — Interphase
DNA replication has occurred. Each chromosome now consists of two identical sister chromatids joined at a centromere.
★ The cell is preparing for division
Phase 1 / 6
Current phase
Interphase
Ploidy
2n
DNA content
4c
Spindle
Not formed
Cells
1
Mitotic index
0%
Bio-time
0 h
▶ Playback
⚙ Cell Type
🎚 Parameters
🎛 Display Toggles
Step labels
Show centromeres
Show spindle
Show centrosomes
Show nuclear envelope
Show chromatid labels
Show ploidy badge
📚 Key fact

Mitosis = nuclear division. The full M phase = mitosis (karyokinesis) + cytokinesis. Each daughter cell receives a genetically identical full set of chromosomes — that's why mitosis is the basis of growth, repair, and asexual reproduction.

2 · The Idea, Step by Step

Scrape your knee and a week later the skin is whole again. Where did the new skin come from? Each cell that was lost is replaced when a neighbouring cell makes a perfect copy of itself and splits in two. That copy-and-split is mitosis. It is how one fertilised egg becomes a whole baby, how a cut heals, and how a strawberry plant sends out runners that grow into new plants. The golden rule is simple: one cell in, two identical cells out — same instructions, no mistakes.

To copy itself cleanly, a cell can't just rip its instructions in half — that would leave each daughter with only part of the manual. So it does the sensible thing first: it makes a full second copy. The instructions live on chromosomes, and before dividing the cell duplicates every one. The two matching copies, called sister chromatids, stay clipped together at a point called the centromere (that's the familiar X-shape). A human cell carries $2n = 46$ chromosomes; after copying it temporarily holds 92 chromatids, then hands a clean set of 46 to each daughter. Copy first, then split — that's the whole trick.

Biologists track the amount of DNA with the symbol $c$, where $1c$ is the DNA in a single (haploid) set. A resting cell sits at $2c$. During S phase the DNA doubles to $4c$. Mitosis then divides that $4c$ evenly, so each daughter ends back at $2c$ — full, not half. The visible division marches through four phases you can scrub through above: Prophase (chromosomes condense), Metaphase (they line up at the equator), Anaphase (sister chromatids are pulled to opposite poles), and Telophase (two new nuclei form), followed by cytokinesis splitting the cell. How often a tissue is caught mid-division is captured by the mitotic index, $\text{MI} = T_M / T_{\text{cycle}}$. For a 24-hour cycle with mitosis lasting about 1 hour, $\text{MI} \approx 1/24 \approx 4\%$ — which is why, at any instant, most cells you look at are not dividing.

Try this in the sim above. Step slowly from Metaphase into Anaphase and watch the moment the joined chromatids let go and slide apart — that single instant is what separates the two phases. Next, switch the cell type to Colchicine-treated: the spindle never forms, so the chromosomes are stranded and the cell stalls — the same trick doctors use to freeze chromosomes for a karyotype. Finally, open the DNA Content Graph tab and follow the line climb from $2c$ to $4c$ during S phase and drop back to $2c$ at cytokinesis — proof that the cell doubles before it halves.

3 · Biological & Mathematical Analysis

The Process — Mitosis

Mitosis — Walther Flemming (1882) first described and named the process from observations of salamander cells; Theodor Boveri & Walter Sutton (1902–1904) showed chromosomes carry hereditary information; Tim Hunt, Lee Hartwell & Paul Nurse (Nobel Prize 2001) discovered the cyclin/CDK control system. Mitosis is the process by which a single eukaryotic cell divides its replicated chromosomes equally into two genetically identical daughter cells. It is the engine of growth (a baby becomes an adult through ~10¹⁴ mitotic divisions), tissue repair (skin, gut lining, blood cells), and asexual reproduction in many organisms.

$$ \text{One parent cell (2n, 4c)} \;\xrightarrow{\text{mitosis + cytokinesis}}\; \text{Two daughter cells (2n, 2c each)} $$

where n = number of chromosome types and c = DNA content multiple (1c = haploid DNA quantity).

Key Structures and Variables

Symbol / StructureBiological meaningWhen visibleA-level relevance
ChromatinLoose DNA + histone proteinsInterphaseNot visible as discrete chromosomes
ChromosomeCondensed chromatin, visible under microscopeProphase onwardsMade of 2 sister chromatids after S phase
Sister chromatidsTwo identical DNA molecules joined at centromereProphase, metaphaseResult of S-phase replication
CentromereConstricted region joining sister chromatidsAll phasesSpindle attachment point
KinetochoreProtein complex on each centromereProphase–anaphaseWhere spindle microtubules attach
Centrosome / Centriole pairMicrotubule-organising centreAlways (one per cell, doubles during S)Sends out spindle fibres
Spindle fibresMicrotubules pulling chromosomesProphase–anaphaseMade of tubulin protein
Metaphase plateImaginary plane at cell equatorMetaphaseWhere all chromosomes align
$2n$Diploid chromosome numberHumans 2n = 46; fruit fly 2n = 8
$c$DNA content (1c = haploid amount)2c → 4c during S; 4c → 2c in anaphase/telophase
MIMitotic index = (cells in mitosis / total) × 100%Measure of how actively a tissue divides

Step-by-step explanation (visualize first, then quantify)

Why does the cell need mitosis? Multi-cellular life is impossible without it. A human starts as one fertilised egg and grows to ~30 trillion cells — every one a descendant of that zygote through mitotic divisions. Every minute, ~300 million of your cells divide to replace lost ones (skin, gut, blood, immune cells). Mitosis must be precise: every daughter must get exactly the right chromosomes.

  1. Interphase — preparation (G1, S, G2). Mitosis is the brief, dramatic act; interphase is the long preparation. In G1 the cell grows and produces proteins. In S phase DNA is replicated — every chromosome now consists of two identical sister chromatids joined at the centromere. In G2 the cell makes the proteins it needs for division (tubulin, cyclins). Total interphase: ~23 hours of a 24-hour cycle.
  2. Prophase — chromosomes condense. Chromatin coils up into visible chromosomes (compaction ratio ~10,000×). The nuclear envelope breaks down into vesicles. Centrosomes (which doubled in S phase) move to opposite poles and start sending out microtubules. The mitotic spindle begins to form.
  3. Metaphase — alignment. Spindle fibres from each pole attach to the kinetochore of every chromosome. Through a tug-of-war of polymerisation and depolymerisation, each chromosome is positioned exactly at the cell's equator — the metaphase plate. The spindle assembly checkpoint prevents progression to anaphase until every kinetochore has correctly attached to both poles.
  4. Anaphase — separation. Once the checkpoint is satisfied, the enzyme separase cleaves the cohesin proteins holding sister chromatids together. Spindle fibres shorten (microtubule depolymerisation), pulling the now-separated sister chromatids to opposite poles. Each chromatid is now a chromosome in its own right. Anaphase is fast — usually under 5 minutes.
  5. Telophase — re-forming nuclei. Chromosomes reach opposite poles and decondense back into chromatin. A new nuclear envelope assembles around each set. The nucleolus reappears. The spindle disassembles. At this point, two complete nuclei exist within one cell.
  6. Cytokinesis — splitting the cytoplasm. In animal cells, a ring of actin and myosin contracts the cell membrane inwards, forming a cleavage furrow that pinches the cell in two. In plant cells, vesicles from the Golgi gather at the equator and fuse to form a cell plate, which grows outwards to make a new cell wall. Mitosis (the nuclear division) and cytokinesis (the cytoplasm division) together complete M phase.

Cell Cycle Mathematics

The cell cycle follows a strict timing. For a typical human somatic cell with a 24-hour cycle:

$$ T_{\text{cycle}} = T_{G_1} + T_S + T_{G_2} + T_M $$

Typical values: $T_{G_1} \approx 11$ h, $T_S \approx 8$ h, $T_{G_2} \approx 4$ h, $T_M \approx 1$ h.

Mitotic Index

A standard A-level technique is counting cells in a stained tissue under a microscope and calculating the mitotic index — the fraction of cells caught in the act of dividing:

$$ \boxed{\;\text{MI} \;=\; \dfrac{\text{number of cells in mitosis (P + M + A + T)}}{\text{total number of cells observed}} \times 100\%\;} $$

For a continuously dividing tissue, the mitotic index is approximately the fraction of cycle time spent in mitosis:

$$ \text{MI} \;\approx\; \dfrac{T_M}{T_{\text{cycle}}} \times 100\% $$

For human gut lining (cycle ~24 h, mitosis ~1 h), MI ≈ 4%. For brain neurons (do not divide), MI ≈ 0%. For cancer cells, MI can exceed 20% — making it a key diagnostic indicator of tumour aggressiveness.

Chromosome Counting Through Phases

For a cell of diploid number $2n$ (humans: $2n = 46$):

PhaseChromosomesChromatidsDNA content
G12n = 462n = 46 (1 per chrom.)2c
After S phase / G2 / Prophase / Metaphase2n = 464n = 92 (2 per chrom.)4c
Anaphase (separation moment)4n = 92 (each chromatid is now a chromosome)4c
Each daughter after telophase + cytokinesis2n = 462n = 462c

What the simulation shows

The default Cell View tab is a top-down view of a single dividing cell. The cell membrane is the outer ring; the nuclear envelope (when present) is the inner ring. Chromosomes are drawn as paired bars — two sister chromatids joined at an orange centromere. Homologous pairs are colour-coded (blue, red, yellow, purple) so you can track which chromatid goes where. The spindle fibres (purple) emerge from two centrosomes (orange) at opposite poles. Watch the chromosomes condense in prophase, line up in metaphase, separate in anaphase, and the cell pinch in two during cytokinesis.

The Cell Cycle Timeline tab shows the proportional durations of G1, S, G2, and M phases. The DNA Content Graph plots how the DNA amount per cell changes through the cycle — flat at 2c during G1, doubling during S to 4c, flat at 4c through G2 and M, then dropping back to 2c at cytokinesis. The Mitosis vs Meiosis tab side-by-side compares the two divisions to emphasise their fundamental differences.

Try the Colchicine-treated preset — colchicine is a real drug that binds tubulin and prevents spindle formation. The cell gets stuck in metaphase with chromosomes free-floating, unable to separate. This is exploited in karyotyping (so you can photograph chromosomes at maximum condensation) and was the basis of early chemotherapy.

Worked numerical example

Example — Mitotic index in a root tip

A student stains an onion root tip and counts cells in 5 microscope fields. The results are:

FieldTotal cellsProphaseMetaphaseAnaphaseTelophase
1523101
2482210
3554102
4503211
5452101
Total25014725

Q1: Calculate the mitotic index.

Cells in mitosis = 14 + 7 + 2 + 5 = 28

$$ \text{MI} = \dfrac{28}{250} \times 100\% = 11.2\% $$

Q2: If total cycle time is 20 hours, estimate the duration of M phase.

$$ T_M = \text{MI} \times T_{\text{cycle}} = 0.112 \times 20\ \text{h} = \mathbf{2.24\ h} $$

Q3: Why is prophase the most-counted phase?

Because prophase lasts longer than the other mitotic phases — chromosome condensation and spindle assembly take time, while anaphase (mechanical separation) is over in minutes. A higher mitotic-index reading for one phase reflects that phase's duration, not its importance. Takeaway: the mitotic index reads time, not significance — and is the standard A-level technique for measuring how actively a tissue is dividing.

References

Campbell, N. A. & Reece, J. B. — Biology, 12th ed., Pearson, 2021, Chapter 12: "The Cell Cycle".
Alberts, B. et al. — Molecular Biology of the Cell, 7th ed., W. W. Norton, 2022, Chapter 17: "The Cell Cycle".
Jones, M., Fosbery, R., Taylor, J. & Gregory, J. — Cambridge International AS & A Level Biology Coursebook, 5th ed., Hodder/CUP, 2022, Chapter 5: "The mitotic cell cycle".
Allott, A. & Mindorff, D. — Biology for the IB Diploma, 3rd ed., OUP, 2023, Topic 1.6.
Flemming, W. — Zellsubstanz, Kern und Zelltheilung, Vogel, 1882 (original description of mitosis).
Nurse, P. — "A long twentieth century of the cell cycle and beyond", Cell, 100: 71–78, 2000.

4 · Frequently Asked Questions

🧬 ConceptualWhy don't the daughter cells have half the DNA each, if the cell just split?
Because the DNA was already doubled before mitosis began. During S phase (in interphase), every chromosome is replicated, so each chromosome consists of two identical sister chromatids. When mitosis starts, the cell has 4c worth of DNA (twice the normal amount). When sister chromatids separate in anaphase and the cell divides in cytokinesis, each daughter gets exactly the original 2c amount. So while it looks like the cell "halved", really the cell first doubled its DNA and then split evenly — the result is two complete sets, not two half-sets. Key takeaway: S phase doubles. Mitosis halves the doubled amount. Net effect: two cells with identical full DNA.
🌍 AppliedWhere does this appear in medicine and biotechnology?
Mitosis is at the heart of cancer biology — every tumour is, by definition, a population of cells dividing when they shouldn't. Most chemotherapy drugs target mitosis: taxanes (paclitaxel/Taxol) stabilise spindle microtubules and prevent disassembly in anaphase; vinca alkaloids (vincristine, vinblastine) prevent spindle assembly entirely; methotrexate blocks DNA synthesis, so cells can never reach mitosis. Karyotyping — the photograph of chromosomes used to diagnose Down syndrome (trisomy 21) and other chromosomal disorders — is done on cells arrested at metaphase using colchicine. Mitotic index is used as a tumour grading criterion in pathology — high MI correlates with aggressive cancer. Stem cell therapy relies on the controlled mitotic proliferation of stem cells in culture before transplantation. Even wound healing, scar formation, and immune response all depend on rapid mitosis in specific cell types. Key takeaway: Cancer is mitosis out of control; chemotherapy is mitosis sabotaged on purpose. Understanding the cycle is the foundation of oncology.
🔬 SimulationWhat exactly is the simulation showing?
The Cell View tab is a top-down cross-section of one dividing cell. The outer cyan ring is the cell membrane. The inner purple ring (present in interphase, prophase start, and telophase) is the nuclear envelope. The coloured bars are chromosomes — each one is drawn as two sister chromatids (the two parallel bars) joined at an orange centromere. Different colours represent different chromosomes from a homologous set (blue/red are one homologous pair, yellow/purple another, etc.). The two orange dots at the poles are centrosomes. The faint purple lines are spindle fibres (microtubules) connecting centrosomes to chromosomes. As you step through the phases, watch the chromosomes condense, align, separate, decondense, and finally the cell pinches in two via the cleavage furrow. Key takeaway: Every step you see is real molecular choreography that happens in your body about 300 million times per minute.
💡 Non-obviousIf mitosis produces identical cells, how do we get different cell types?
All cells in your body have the same DNA, but they are not all the same. The difference is in which genes are expressed. A liver cell and a neuron carry the same 23,000 genes, but the liver cell turns on liver-specific genes (albumin synthesis, urea cycle enzymes), while the neuron turns on neuron-specific genes (ion channels, neurotransmitter synthesis). This is called differential gene expression, controlled by transcription factors and epigenetic marks (DNA methylation, histone modification). When a liver cell divides by mitosis, it produces two liver cells — not because it gives "liver-specific DNA" but because the epigenetic pattern is also inherited. So mitosis is genetically conservative but epigenetically can pass on cell identity. Key takeaway: Same DNA, different active genes. Differentiation is about which genes are switched on, not which genes are present.
📐 QuantitativeHow is the mitotic index used in practice?
A pathologist examining a tumour biopsy counts cells in mitosis under high magnification (usually 10 high-power fields) and reports the mitotic count. For breast cancer, the Nottingham Grading System assigns 1–3 mitotic points (1 = <7 mitoses per 10 fields, 3 = ≥18) that contribute to the overall grade. Higher MI = more rapid tumour growth = generally worse prognosis but also more responsive to chemotherapy (because cytotoxic drugs work best on dividing cells). For A-level practical work, you'd stain an onion root tip with acetic orcein, count cells in 4–5 fields, divide cells in mitosis by total cells, and multiply by 100. From the MI you can back-calculate the duration of mitosis if you know the cycle length, using $T_M = \text{MI} \times T_{\text{cycle}}$. Key takeaway: MI is both a clinical diagnostic tool and an A-level practical. It reads how busy a tissue is dividing.
🎓 DeepWhat happens when mitosis goes wrong?
Errors in mitosis are the root of two huge problems. First, cancer: when the cell cycle checkpoints fail (mutations in p53, RB, BRCA1, or APC genes), cells divide when they shouldn't, leading to tumour formation. The "guardian of the genome", p53, is mutated in over half of all human cancers. Second, aneuploidy: if chromosomes fail to separate correctly in anaphase (called non-disjunction), daughter cells end up with the wrong chromosome count. In gametes this causes conditions like Down syndrome (trisomy 21 — three copies of chromosome 21); in somatic cells it's a hallmark of cancer cells, which routinely have 60–80 chromosomes instead of 46. Mitotic catastrophe — when a cell tries to divide with severe DNA damage — usually triggers programmed cell death (apoptosis), serving as a tumour-suppressor mechanism. Drugs like olaparib (PARP inhibitor) push cancer cells with defective DNA repair into mitotic catastrophe selectively. Key takeaway: Mitotic errors are the molecular root of both cancer and chromosomal disease — and modern therapy exploits both.
🧬 ConceptualWhat's the difference between mitosis and cytokinesis?
Mitosis is the division of the nucleus — the prophase, metaphase, anaphase, telophase sequence where chromosomes are divided into two sets. Cytokinesis is the division of the cytoplasm — the physical splitting of the cell into two daughter cells. They are separate events: in some cells (early fruit fly embryos, fungal hyphae), mitosis occurs many times without cytokinesis, producing one giant cell with many nuclei (a syncytium). Conversely, cytokinesis without mitosis is impossible — there must be two nuclei first. Together, mitosis + cytokinesis = M phase of the cell cycle. The mechanisms also differ: mitosis uses microtubules (spindle), while cytokinesis uses actin/myosin (contractile ring) in animals or vesicle fusion (cell plate) in plants. Key takeaway: Mitosis splits the nucleus, cytokinesis splits the cell. M phase = both together.

Further resources

HHMI BioInteractive — "Mitosis" animations (biointeractive.org)
Khan Academy Biology — "Cell Cycle & Mitosis" (khanacademy.org/science/ap-biology/cell-communication-and-cell-cycle)
Bozeman Science — "Mitosis" (youtube.com/@bozemanscience)
Amoeba Sisters — "Mitosis: The Amazing Cell Process" (youtube.com/@amoebasisters)
Crash Course Biology Ep. 12 — "Mitosis: Splitting Up is Complicated" (youtube.com/@crashcourse)

5 · Misconceptions & Common Errors

A · Conceptual misconceptions

❌ Misconception: "Sister chromatids separate in metaphase."

✅ Correction: Sister chromatids separate in anaphase, not metaphase. In metaphase, they are aligned at the equator, still held together at the centromere by cohesin protein. Only at the metaphase-to-anaphase transition does the enzyme separase cleave cohesin, and the spindle fibres then pull the now-separated chromatids to opposite poles. Mixing up metaphase (alignment) with anaphase (separation) is the most common diagram-labelling error in exams.

📖 Campbell & Reece, Biology, 12th ed., Ch. 12, Figure 12.7; Alberts, Ch. 17.5.

❌ Misconception: "Each daughter cell after mitosis has half the DNA of the parent."

✅ Correction: Each daughter cell has the same amount of DNA as the original G1 parent (2c) — and genetically identical to it. The confusion arises because students forget that DNA was doubled during S phase before mitosis. The parent cell at the start of mitosis had 4c (doubled); mitosis splits 4c into two 2c daughters. Saying "halved" is correct if you mean "halved from the 4c at metaphase", but wrong if you compare daughter to G1 parent — they are equal.

📖 Jones et al., Cambridge International AS & A Level Biology, 5th ed., Ch. 5.

❌ Misconception: "Mitosis only happens during growth."

✅ Correction: Mitosis happens throughout your entire life — to replace lost cells. Your gut lining is completely renewed every 5–7 days; red blood cells live ~120 days and are replaced at ~2 million per second; skin cells slough off and are replaced constantly. Even adult tissues like liver retain the capacity for mitosis after injury. The only major exceptions are neurons (mostly post-mitotic in adults) and cardiac muscle cells (very limited turnover). For most of your body, mitosis is a continuous, life-long process — not just for growing up.

📖 Alberts et al., Molecular Biology of the Cell, 7th ed., Ch. 22 "Stem Cells and Tissue Renewal".

B · Common exam & calculation errors

❌ Error: Drawing chromosomes as X-shapes during interphase.

✅ Correct approach: During G1 interphase, each chromosome is a single DNA molecule — drawn as one bar, not an X. The X-shape only appears after S phase, when DNA has replicated to give two sister chromatids joined at a centromere. So G1 = single bars; G2 + prophase + metaphase = X-shapes; after anaphase separation = single bars again. Examiners deduct marks when students draw chromosomes consistently as X-shapes regardless of phase.

🔍 Textbook diagrams show condensed chromosomes (which are X-shaped post-S phase) so students assume that's the universal shape.

❌ Error: Computing mitotic index without recognising that prophase is over-represented because it lasts longer.

✅ Correct approach: The fraction of cells in each phase is proportional to the phase's duration, not its biological importance. If you see 4× more prophase cells than anaphase cells, prophase lasts ~4× longer than anaphase — that's the standard interpretation. To estimate the duration of any phase: $T_{\text{phase}} = \dfrac{\text{cells in phase}}{\text{total cells in mitosis}} \times T_M$. This is a classic A-level practical calculation worth knowing.

🔍 Students confuse cell count with reaction rate — but in a steady-state population, count reflects time spent.

❌ Error: Writing "mitosis produces gametes."

✅ Correct approach: Mitosis produces somatic (body) cells — identical, diploid daughter cells used for growth and repair. Gametes (sperm and egg) are produced by meiosis in the gonads, which halves the chromosome number and produces four genetically unique haploid cells. Confusing mitosis with meiosis is the single most common exam mistake in cell-division questions. Always check: identical or different? diploid or haploid? somatic or gamete?

🔍 Both processes start similarly and use the same terminology (prophase, metaphase, etc.), making them easy to mix up.

References

Campbell, N. A. & Reece, J. B. — Biology, 12th ed., Pearson, 2021, Ch. 12.
Alberts, B. et al. — Molecular Biology of the Cell, 7th ed., W. W. Norton, 2022, Ch. 17.
Jones, M. et al. — Cambridge International AS & A Level Biology Coursebook, 5th ed., 2022, Ch. 5.
Cambridge International Examiners' Reports (9700 Biology Paper 2 & 4) — recurring errors in chromatid separation timing and mitotic-index calculation.
AQA A-level Biology 7402 Examiners' Report — Topic 3.4 "Cell cycle": chromosome shape and meiosis-vs-mitosis confusion.